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Our laboratory demonstrated that SERT and αvβ3 receptors colocalize in midbrain presynaptic terminals (Mazalouskas et al., 2015), suggesting that SERT is localized to multiprotein complexes coordinated by integrin αvβ3 in midbrain synapses. Prior scientific studies suggest that in platelets, the association of SERT with activated αIIbβthree integrins, and its localization to adhesion complexes, are correlated with improved uptake activity (Carneiro et al.
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3 integrin is activated by talin binding on activation of inside of-out signaling, represented listed here by PAR4 activation. Talin binding separates the intracellular domains of α
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three, amplified mobile adhesion, without any consequences on platelet aggregation in vitro. This mutation has spectacular results on platelet dimension and is particularly present in Glanzmann thrombasthenia individuals, suggesting that constitutive activation of α
3 ligand-binding area had been assayed utilizing an antibody that recognizes the Energetic conformation of α
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